Papers: 10.1371/journal.pgen.1008217

https://doi.org/10.1371/journal.pgen.1008217

A mutant form of Dmc1 that bypasses the requirement for accessory protein Mei5-Sae3 reveals independent activities of Mei5-Sae3 and Rad51 in Dmc1 filament stability

Cited by: 8
Author(s): Diedre Reitz, Jennifer Grubb, Douglas K. Bishop
Published: almost 7 years ago

Software Mentions 2